WTS 13-Zymobac GTLS Treatment Dramatically Reduces Hydrogen Sulphide and Odours In An Industrial Waste Water System
Summary
Hydrogen Sulphide (H2S) was reduced on average by >96% from the top hatch of the balance tank of an industrial waste water treatment system after daily treatment with WTS 13-Zymobac GTLS.
Background
An industrial waste disposal and treatment site located in North Queensland receives an average of 26 kL/day of various waste water streams into an aerated balance tank. These waste waters include septic, industrial oils, fats and greases, as well as waste chemicals. H2S levels from the 300kL balance tank averaged 553 mg/L, with spikes of up to 670 mg/L of H2S occurring regularly. These high levels of H2S are the cause of odour complaints from local housing developments, and also give rise to safety and corrosion concerns at the site.
Objectives
The treatment objective was to reduce the H2S and odours being discharged from the vessel, reducing odours to the site and surrounding areas.
Method
WTS 13-Zymobac GTLS was manually administered to the balance tank. An initial dose of 200 mL was added, and after a period of 6 days an ongoing dose of 200 mL was added every day by the site operators. To demonstrate success, H2S concentrations were monitored with an odour logger placed at the top hatch of the tank, with values collected for 5 days before the initial dose, and H2S logging ongoing throughout the trial. H2S values were recorded every 5 minutes, with the results of the trial shown in Figure 1.
Results
After the initial WTS 13-Zymobac GTLS dose of 200 mL, H2S values plummeted to an average of 104 mg/L, with peaks reduced to less than 300 mg/L of H2S.
Ongoing daily dosing of WTS 13-Zymobac GTLS reduced H2S levels even further, with most daily H2S peaks less than 20 mg/L. Occasional high spikes from large influent flows quickly reduced back to sub 20 mg/L levels in less than an hour.
The reduction in H2S levels and odours has persisted for the period of WTS 13-Zymobac GTLS treatment, with H2S levels returning to the previous high levels when the treatment is halted.
